A MTW lunch seminar with Michael Schöll, now home from Berkeley
Two harmful proteins that accumulate in the brains of Alzheimer’s disease (AD) patients are β-amyloid (Aβ) as amyloid plaques, and hyperphosphorylated forms of the microtubule-associated protein tau assembling into neurofibrillary tangles (NFT). Neuropathological studies have repeatedly demonstrated that these proteins also accumulate in the brains of cognitively healthy older people.
Tau rather than Aβ pathology has been shown to be closely related to cognitive impairment, predominantly memory decline, in post mortem studies in both aging and AD. The advent of positron emission tomography (PET) tau ligands enables, for the first time, tau pathology in living humans to be topographically mapped, quantified, and examined in relation to other pathology markers and aspects of cognitive decline.
At this seminar, assistant professor Michael Schöll will present results from his research conducted during the past year as a visiting scholar at the University of California, Berkeley, where he examined uptake patterns of a tau PET ligand in the brains of young and elderly healthy subjects as well as in patients with AD.
Michael Schöll is an assistant professor at the Department of Clinical Neuroscience and Rehabilitation at the University of Gothenburg and MedTech West.
A light lunch will be served to those who have registered on November 26. Welcome!
Date: Dec 2, 2015
Time: 11:30-12:30 (13:00 incl. lunch)
Place: Kammaren, Vita stråket 12, Sahlgrenska University Hospital
(If you have any problems registering below, please email: email@example.com).